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They also modulate the action of antioxidant enzymes

Ferroptosis is morphologically, biochemically, and genetically distinct from necrosis, apoptosis, and autophagy, and these differing features include abnormal mitochondrial membrane density, iron accumulation, lipid peroxidation, overexpression of ferroptosis biomarkers, and death of leucocyte subsets and the corresponding loss of immune function [11, 18,19,20,21]

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the sulphydryl and -glutamyl groups) it may to some extent compensate for glutathione in the cellular defence against oxidative stress

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